![]() ![]() However, in the RE group, any alterations in responsiveness to NP and NS were unchanged during PEH compared with preexercise. In the non-RE group, the responsiveness of CBR-mediated changes in HR, MAP, and vascular conductance increased, particularly in response to −40 mmHg NS during postexercise compared with preexercise. The other eight participants did not display a reduction in MAP after exercise (non-RE group). Eight participants showed substantial postexercise hypotension (PEH) during recovery from exercise (Δ MAP: approximately −5 ± 0.9 mmHg, P < 0.05). To determine peripheral circulation, vascular conductance in active and inactive limbs was measured. As the central hemodynamics measurements, heart rate (HR), mean arterial pressure (MAP), cardiac output, and total vascular conductance were assessed. Both pre- and postexercise, the CBR-mediated responses of the central and peripheral hemodynamics were evaluated using 5-s periods of NP and NS (−60, −40, or +40 mmHg). Sixteen healthy young male participants (age: 27 ± 1.5 yr) were in a supine position for 30 min preexercise, followed by 60 min of cycling exercise, and then returned to a supine position for an additional 60 min postexercise. This study aimed to examine the carotid baroreflex (CBR) control of the central and peripheral hemodynamics after exercise using the neck pressure (NP) and neck suction (NS) technique. ![]()
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